Field studies were conducted in 2000 and 2001 in Plains, GA, to determine peanut and weed response to the residual herbicides sulfentrazone, imazapic, diclosulam, and flumioxazin. Herbicide treatments included sulfentrazone applied preemergence (PRE) or preplant incorporated (PPI) at 112, 168, 224, and 280 g ai/ha, imazapic postemergence (POST) at 71 g ai/ha, diclosulam PPI at 26 g ai/ha, and flumioxazin PRE at 88 g ai/ha. Peanut exhibited early-season injury from all herbicide treatments, ranging from 0 to 10% for sulfentrazone PPI or PRE, 10% for imazapic, 3 to 23% for flumioxazin, and 1 to 7% for diclosulam. Yields were similar for sulfentrazone PPI- or PRE-treated and flumioxazin-, imazapic-, and diclosulam-treated peanut. Yellow nutsedge control was 83% or greater with all rates of sulfentrazone PRE or PPI, 83 and 90% with diclosulam, and 96 and 99% with imazapic, respectively. Flumioxazin did not control yellow nutsedge or wild poinsettia. Tall morningglory control was 82% or greater with imazapic, diclosulam, flumioxazin, and sulfentrazone PPI or PRE at 168 g/ha or higher. Florida beggarweed control was 88% or greater with diclosulam, flumioxazin, and sulfentrazone PRE at 224 and 280 g/ha. Overall, peanut tolerance to sulfentrazone at 112 to 280 g/ha PPI and PRE was high and yield was equivalent to the currently registered peanut residual herbicides.
Nomenclature: Diclosulam; flumioxazin; imazapic; sulfentrazone; Florida beggarweed, Desmodium tortuosum (Sec) L., #3 DEDTO; tall morningglory, Ipomoea purpurea (L.) Roth # PHBPU; wild poinsettia, Euphorbia heterophylla L. # EPHHL; Yellow nutsedge, Cyperus esculentus L. # CYPES; peanut, Arachis hypogaea L.
Additional index words: Peanut injury, peanut yield, susceptibility to herbicides.
Abbreviations: ALS, acetolactate synthase; POST, postemergence; PPI, preplant incorporated; PRE, preemergence; VE, vegetative emergence.
